If the whole point is to control insulin levels while on CNS, then why is it perfectly fine to take leucine and the new carb shock while in CNS?

Have you read the book?

If the whole point is to control insulin levels while on CNS, then why is it perfectly fine to take leucine and the new carb shock while in CNS?

Have you read the book?

Better yet, listened or read ANY of the new material that has been released lately?

My impression was that you'd be controlling insulin in order for you to reach ketosis.. if not.. then what energy are you truly tapping into?

The good quality fats you're consuming. That is what you're using for fuel, that's the whole premise of eating ULC. Ketogenic diets are different. Again, a pressing medical condition or overly obese would call for different measures in nutrition. As such would be a Keto diet, where you're eating more fats than protein. Your body will also be using body fat as the fuel source, not carbs. To revamp your metabolism and get hormones churning once again is why there is a refeed. Some folks can/need to go longer, so do not.

I'm not really sure what Stephanie Person is trying to prove by all of her ketogenic diet videos.  One of her videos show pictures of her before she started her ketogenic diet and after.  She was ripped before, and she's still ripped after.  I guess I just don't get how her diet has made her more healthy when she was clearly healthy and ripped before she even started the diet. 

My big problem with going on ketogenic diet for as long as stephanie has.. is if someone is obese, and they are losing too much fat, they might get into ketoacidosis. Because the body is producing toxic levels of acid to burning the abdunance of fat. I think stephanie never needed to worry about it because she never had that abundant supply of body fat to convert to ketones, but if someone who is obese is following her, they might run into a risk of too much ketones. Which is why I think carb nite is a safer alternative.Secondly, people might be losing way too much electrolytes due to a harsh balance of potassium, magnesium, and sodium.. which might be found in vegetables.. but this has been a huge problem for people who have been on ketogenic diets for a long period of time. People on ketogenic diets needs a higher amount of potassium and magnesium in their system.However, carb nite addresses these problems as it introduces these carb cyclic refeeds.As far as my last quesiton is concerned of when the body starts burning body fat as opposed to consumable fat. It's when you stop eating and enter a fasting state. Your body eats consumable fats to increase fatty acids to break down into keytones. Your body now prefers keytones instead of glucose and amino acids.. so when your body stops eating, then it starts grabbing energy from your body fat. But like I said.. if someone is obese.. and too much acid and ketones is taken out.. it would be really hazardous.

My big problem with going on ketogenic diet for as long as stephanie has.. is if someone is obese, and they are losing too much fat, they might get into ketoacidosis.

This is just false.Ketoacidosis only occurs in people who are diabetic.  It happens when BOTH blood glucose AND blood ketones are high.There is also a form of ketoacidosis that involves alcoholics, but that does not pertain to this discussion.I highly recommend to anyone out there who wants to have a knowledgeable conversation about ketosis:  Read Jimmy Moore's book.  And/or read the work of Volek & Phinney, where Jimmy got a lot of his info.

Thanks. I was reading incorrect info from a dietician on a diabetic website. Literally stating.. and I quote... "Burning too much fat can result in a dangerous condition called ketoacidosis, which means too much acid in your blood". I usually avoid these articles.. but I sometimes read on them as they explain things for the layman much better than reading an extensive study (which I normally do). One thing though, ketoacidosis doesn't matter on whether glucose is present or not, it's when there's a large amount of keytones present and there's not enough insulin to regulate the flow of the elevated fatty acids and the production of ketone bodies. If glucose was a factor, then it wouldn't be called "ketoacidosis".

One thing though, ketoacidosis doesn't matter on whether glucose is present or not, it's when there's a large amount of keytones present and there's not enough insulin to regulate the flow of the elevated fatty acids and the production of ketone bodies. If glucose was a factor, then it wouldn't be called "ketoacidosis".

I'm sorry, but, again -- false.  Just... FALSE.  🙁Remember, we are talking about people who are diabetic here.  Generally Type 1, or Type 2 folks who are truly insulin-dependent.This occurs when they are not regulating their insulin well enough.You yourself just said:  "there's not enough insulin".  If there is not enough insulin for a diabetic person... what happens?I hope I can clear this up once and for all.From Jimmy's book:"When diabetics do not get an adequate amount of insulin, their bodies respond as if they are starving.  Their bodies think there's no more glucose to be had, either from diet or glycogen stores, and they switch to burning fat instead and ramp up ketone production so it can be used as an alternative energy source.  The problem is, these diabetics aren't out of glucose -- in fact, they have elevated levels of blood glucose.  Insulin is the hormone that allows glucose into cells, and without it, the blood sugar has nowhere to go and accumulates in the bloodstream, even as the body can't stop making ketones."

Okay so what happens when the blood glucose levels drop and you are out of insulin, but you can't stop making ketones and control the flow of fatty acids? That state would still be considered as Ketoacidosis.. and apparently.. this has occured with people who are diabetic and pregnant.http://www.ncbi.nlm.nih.gov/pubmed/18588610There have been cases when alcohol can induce ketoacidosis, but leave the person in a state of hypoglycemia.. because of the alcohol that was taken:http://www.ncbi.nlm.nih.gov/pubmed/12444383In both of these case studys, they were both defined as being in a state of Ketoacidosis. The proper definition does not include the levels of glucose, but is typically used as an indicator in most circumstances.

I don't have a point, nor a basis or argument here. I'm trying to formulate a discussion here to get more truths and answers.. and thinking critically on this sort of information.plus I've been agreeing with you on all points.. thanks to you reading the book by Jimmy, I started to do more research of my own.. verifying everything you pointed out and then some! But only disagreement is the blood glucose levels in ketoacidosis:I'm referring to the studies of glucose because it's proof that glucose has nothing to do with ketoacidosis. You can get into what's called Euglycaemic diabetic ketoacidosis, which means ketoacidosis with people with normal to low blood glucose levels. YOU are referring to ketoacidosis for people who have relatively high blood glucose levels. Like I said.. anyone with diabetes type 1 can have ketoacidosis regardless of glucose level. I'm not wrong on this one. So no more referring to another one of Jimmy's quotes.. because obviously Jimmy doesn't know about this form of ketoacidosis.I'm not a doctor.. so I am less liable.. therefore can afford to make mistakes and admit to them