You may be taking in too much protein. If I remember correctly the liver will break down protein and turn it into glucose if your fat intake isn't high enough.

Do not wish to confound but keep an eye on your sleep.  I changed nothing diet/training wise and found huge variations in my AM glucose depending on how sleep-deprived I was.I tend to need about 6.5 hours or a bit longer if training but there is quite a bit of individual variation so you will likely be different.

I have found something this kind of information:http://high-fat-nutrition.blogspot.de/2007/10/physiological-insulin-resistance.html

Very interesting.  Thanks for sharing.

Curious and other friends, Does that mean that CN and ketogenic diets can cause diabetes? according to the article?

Oh, my goodness, NO.  I will be back later if you'd like to discuss further -- but -- just, no.  Keeping insulin levels low, as you do on a ketogenic diet or CN, is ideal for treating and/or preventing diabetes.

Eidah, I went back and read the article again and I read through MANY of the comments at the bottom.  One person specifically asked about Carb Nite, and Peter's response was good.  Too much for me to delve into at this point…  But if you are interested in learning more, I recommend reading through the comments on that blog post you are talking about.  Lots of clarification and deep thinking going on there.I am in the middle of reading Phinney & Volek's The Art and Science of Low Carbohydrate Living right now, and I can just tell you that it is very scientific, it goes into great detail about insulin resistance, metabolic syndrome, etc.  A great read if you are up for it.  If I find some good snippets I can quote here for you, I'll try to come back and do that.  But really, if you want to KNOW what you are talking about, I say get it right from the experts who have made it their life's work to research this stuff.In short, I will just say that my mother has been diagnosed with insulin resistance, she knows she is on the road to Type 2 diabetes, she is now on Metformin, and I cannot tell you how much I wish she would go ULC.  These diets are the answer to these problems -- not the cause. 

Eidah, someone posted this on another forum this morning and I thought it may be helpful for you to read, also:http://high-fat-nutrition.blogspot.com/2008/08/physiological-insulin-resistance.htmlI think it is key for you to understand the difference between physiological insulin resistance and pathological insulin resistance.Folks on Peter's blog seem to agree that this is an overly-simplistic summary, but perhaps that is what you need, if you don't want to get into the (very deep) details:  (Copied and pasted from the comments section of above link -- bolding mine):"Dr. Michael Eades (a long-time LC proponent) suggests that the high FBG in low-carb eaters has to do with a lack of enzymes:It’s not really the “physiologic insulin resistance from fat” that causes the problem you describe; it is simply a temporary deficiency of enzymes. Virtually all physiological functions in the body are controlled by the enzymes that catalyze the various reactions required. Our DNA codes for all of these enzymes, all of which are proteins. Our DNA codes for many enzymes that we may never use because we don’t need them given our lifestyle, but the coding and ability to make them are still present. We send signals to the DNA that we need them when we need them, and our RNA/DNA system cranks them out. Such is the case with the enzymes we use to metabolize carbohydrates, which are different than those used to metabolize fats. If we are on a long-term low-carb diet, we don’t need the enzymes to metabolize a lot of carb, so we don’t make them. We do need the enzymes to metabolize fat, so we have plenty of those. If we take in a large load of carbohydrate, we don’t have the enzymes we need to dispose of the carbs quickly, so they remain in the blood as sugar a little longer, giving a glucose tolerance test reading that looks like that of one with glucose intolerance or outright diabetes. If we eat carbs for a few days before, we crank out the enzymes, which are then in place when we take the glucose tolerance test, and we get a normal reading. It has nothing to do with the fat causing insulin resistance; it’s simply a matter of lack of enzymes."